The evolution of spastic pathologies as a consequence of brain damage is a complex phenomenon involving disuse, immobility and brain tissue remodeling [1]. The vicious circle leading to a worsening of the patients’ status proceeds through muscle shortening by contractures, disruption of the normal reflex behaviour and sensory disturbances. A way to prevent chronicity of major consequences could be to favour mobility and any residual use of the affected limb.

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